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Cancer Headlines Researchers at Emory University School of Medicine and their colleagues have discovered a genetic mechanism that controls cellular growth in the fruit fly Drosophila melanogaster, and believe it likely that a similar system may be at work in normal and cancerous human cells. The findings appear in the November issue of the journal Developmental Cell. Ken Moberg, PhD, assistant professor of cell biology at Emory University School of Medicine, is the lead and corresponding author of the paper. Moberg joined the Emory faculty in 2003, and most of the work described in the paper was done in his Emory lab. The senior author is Iswar K. Hariharan, professor of cell and developmental biology at the University of California, Berkeley. The Emory and Berkeley researchers have uncovered important details about how mutational inactivation of the Drosophila version of Tumor Susceptibility Gene 101 (Tsg 101) causes cells to overgrow, leading to organ hypertrophy and tumor-like growths. Scientists first identified the human Tsg101 gene in the mid-1990s based on its ability to control the growth of cells in a culture dish, but little has been learned since then about how it does this. "The work that was done ten years ago strongly implicated Tsg101 as a growth regulatory gene, but how it works has remained largely obscure," Dr. Moberg says.	Cancer Research Prevent Cancer: Why Bother? By Judy Ford Cancer is a word that strikes fear in the heart of individuals and families. When it strikes close to us we sit impotently by as we watch our loved one suffer. The victim is usually offered surgery, radiotherapy and chemotherapy and goes through a dreadful time of illness and suffering, not knowing whether they are winning the battle. Cancers vary. Some are more aggressive than others. Some have a reasonable chance of survival. Other cancers are outright killers. A cancer death is always ugly, usually painful and often degrading. We know that the rates of most cancers are rising, especially breast cancer in women and prostate cancer in men. We know that the death rates in these cancers have dropped but the suffering has not. We also know that a great deal of research has been undertaken but most of the results we hear about are concerned with treatment. Perhaps there is a new wonder drug on the market or a new discovery about how to administer radiation. This research is of course very helpful once we are ill but why isn’t there more effort put into prevention? Do we need to get ill in the first place? There are several fairly obvious reasons why most money, effort and publicity is put into cure rather than prevention. It is really very easy to understand but this bias is hard to justify. First, almost all of us live in a capitalist economy. Businesses have to make money and so they need to sell products. Drug companies invest heavily in each drug that they develop and many never reach the market so pharmaceutical companies need to market their products very strongly in order to recoup money for their shareholders. Second, prevention doesn’t sell newspapers and is hard to prove. The absence of something is never as interesting or startling as drama. Prevention doesn’t captivate the audience in the same way as dramatic cures. Being saved from a trauma is much more interesting than the absence of trauma in the first place. Drama sells newspapers, books, radio, TV and films. Third, there are vested interests in many of the products that are causing cancer. In 1939 there was definite proof that tobacco smoking causes lung cancer. Serious efforts to curtail smoking have only occurred in some countries recently. In others, it hasn’t started. We have a long way to go before we restrict other cancer causing agents. Fourth, politicians don’t win votes on prevention. Prevention is slow. The results are only seen over the long term. Politicians are generally disinterested in things that can’t be accomplished within their term of office. The good news is that a great deal is known about the cancer process and the variety of things that can affect it. There are three major steps in development. They often occur over long periods of time, even decades, but can occur simultaneously in one part of the body. The steps are MUTATION, CANCER PROMOTION and CANCER PROGRESSION. The chances of any of these steps taking place in your body can be significantly reduced by understanding and knowledge. Research has explained most of the mechanisms that underlie cellular errors and the progression of changes from a normal cell to a cancer cell. Intelligent people are capable of understanding these processes even without a Science or Health background. Research has also gone a long way to showing us how the three stages can be eliminated or minimized. This is still work in progress but there is already a great deal that you can do right NOW. Dr Judy Ford is an internationally respected geneticist who has undertaken considerable research into the causes of cancer , and the steps that can be undertaken to prevent cancer or greatly reduce personal risk. Dr Ford worked for 30 years in Cancer Research, specializing in how genetic material can be damaged by some chemicals and radiations. She also contributed a great deal to knowledge about how cells misdivide. She now works as a Science Communicator, explaining Science in lay terms and showing people how they can modify their lifestyles to reduce their risks of cancer. She has recently written an E-Book: Know Your Enemy: Understand and Reduce Cancer Risk. This article may be reproduced in full as long as the author's name and all links remain attached to it. Article Source: http://EzineArticles.com/ DISCLAIMER - Please note that all articles on this web site does not constitute professional advice. All articles are intended to provide a general view of many topical subjects from a variety of sources. We are not responsible for the content or any sponsored links that you may choose to visit from this web site. We suggest you to consult a solicitor and your doctor for advice relevant to you own situation before making any important decisions.  The author is not an expert in any given field. By printing, downloading, or using you agree to our full terms. 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